The insulinotropic effect of endothelin-1 is mediated by glucagon release from the islet alpha cells

Research output: Contribution to journal/Conference contribution in journal/Contribution to newspaperJournal articleResearchpeer-review

Standard

The insulinotropic effect of endothelin-1 is mediated by glucagon release from the islet alpha cells. / Brock, B; Gregersen, S; Kristensen, K; Thomsen, J L; Buschard, Karen Kaas; Kofod, Hans; Hermansen, K; Kristensen, Kurt.

In: Diabetologia, Vol. 42, No. 11, 1999, p. 1302-7.

Research output: Contribution to journal/Conference contribution in journal/Contribution to newspaperJournal articleResearchpeer-review

Harvard

Brock, B, Gregersen, S, Kristensen, K, Thomsen, JL, Buschard, KK, Kofod, H, Hermansen, K & Kristensen, K 1999, 'The insulinotropic effect of endothelin-1 is mediated by glucagon release from the islet alpha cells', Diabetologia, vol. 42, no. 11, pp. 1302-7. https://doi.org/10.1007/s001250051442

APA

Brock, B., Gregersen, S., Kristensen, K., Thomsen, J. L., Buschard, K. K., Kofod, H., Hermansen, K., & Kristensen, K. (1999). The insulinotropic effect of endothelin-1 is mediated by glucagon release from the islet alpha cells. Diabetologia, 42(11), 1302-7. https://doi.org/10.1007/s001250051442

CBE

MLA

Vancouver

Brock B, Gregersen S, Kristensen K, Thomsen JL, Buschard KK, Kofod H et al. The insulinotropic effect of endothelin-1 is mediated by glucagon release from the islet alpha cells. Diabetologia. 1999;42(11):1302-7. https://doi.org/10.1007/s001250051442

Author

Brock, B ; Gregersen, S ; Kristensen, K ; Thomsen, J L ; Buschard, Karen Kaas ; Kofod, Hans ; Hermansen, K ; Kristensen, Kurt. / The insulinotropic effect of endothelin-1 is mediated by glucagon release from the islet alpha cells. In: Diabetologia. 1999 ; Vol. 42, No. 11. pp. 1302-7.

Bibtex

@article{a22dd5d007fb456aa2915e4b866ba044,
title = "The insulinotropic effect of endothelin-1 is mediated by glucagon release from the islet alpha cells",
abstract = "The circulating concentrations of endothelin-1 (ET-1), a peptide derived from endothelium, are increased in hypertension and diabetes. Endothelin-1 has recently been shown to be an insulinotropic agent. The mechanism of action of endothelin-1 on the endocrine pancreas has not yet been clarified.",
keywords = "Animals, Cell Line, Endothelin-1, Glucagon, Insulin, Islets of Langerhans, Male, RNA, Messenger, Rats, Rats, Inbred Lew, Receptor, Endothelin A, Receptors, Endothelin, Reverse Transcriptase Polymerase Chain Reaction",
author = "B Brock and S Gregersen and K Kristensen and Thomsen, {J L} and Buschard, {Karen Kaas} and Hans Kofod and K Hermansen and Kurt Kristensen",
year = "1999",
doi = "10.1007/s001250051442",
language = "English",
volume = "42",
pages = "1302--7",
journal = "Diabetologia",
issn = "0012-186X",
publisher = "Springer",
number = "11",

}

RIS

TY - JOUR

T1 - The insulinotropic effect of endothelin-1 is mediated by glucagon release from the islet alpha cells

AU - Brock, B

AU - Gregersen, S

AU - Kristensen, K

AU - Thomsen, J L

AU - Buschard, Karen Kaas

AU - Kofod, Hans

AU - Hermansen, K

AU - Kristensen, Kurt

PY - 1999

Y1 - 1999

N2 - The circulating concentrations of endothelin-1 (ET-1), a peptide derived from endothelium, are increased in hypertension and diabetes. Endothelin-1 has recently been shown to be an insulinotropic agent. The mechanism of action of endothelin-1 on the endocrine pancreas has not yet been clarified.

AB - The circulating concentrations of endothelin-1 (ET-1), a peptide derived from endothelium, are increased in hypertension and diabetes. Endothelin-1 has recently been shown to be an insulinotropic agent. The mechanism of action of endothelin-1 on the endocrine pancreas has not yet been clarified.

KW - Animals

KW - Cell Line

KW - Endothelin-1

KW - Glucagon

KW - Insulin

KW - Islets of Langerhans

KW - Male

KW - RNA, Messenger

KW - Rats

KW - Rats, Inbred Lew

KW - Receptor, Endothelin A

KW - Receptors, Endothelin

KW - Reverse Transcriptase Polymerase Chain Reaction

U2 - 10.1007/s001250051442

DO - 10.1007/s001250051442

M3 - Journal article

C2 - 10550413

VL - 42

SP - 1302

EP - 1307

JO - Diabetologia

JF - Diabetologia

SN - 0012-186X

IS - 11

ER -