Sustained overexpression of neuropeptide S in the amygdala reduces anxiety-like behavior in rats

Sandra Tillmann, Heidi E. Skibdal, Søren H. Christiansen, Casper R. Gøtzsche, Moustapha Hassan, Aleksander A. Mathé, Gregers Wegener*, David P.D. Woldbye

*Corresponding author for this work

Research output: Contribution to journal/Conference contribution in journal/Contribution to newspaperJournal articleResearchpeer-review

8 Citations (Scopus)

Abstract

Neuropeptide S (NPS) has shown anxiolytic-like effects in rodents after acute administration, but its long-term effects remain unknown. Gene therapy enables the targeted delivery of DNA to cell nuclei, and recombinant adeno-associated viral (rAAV) vectors have been identified as suitable tools for stable overexpression. Thus, to explore the effects of long-term expression of NPS, the present study examined anxiety- and depressive-like effects after rAAV-mediated NPS overexpression in the rat amygdala. Compared to rats injected with an empty control vector (rAAV-Empty), rAAV-NPS treatment was associated with reduced anxiety-like behavior in the elevated plus maze and light-dark box, but did not affect depressive-like behavior in the forced swim test. Importantly, rAAV-NPS did not cause confounding effects on locomotion or bodyweight as opposed to currently used anxiolytic drugs. Immunohistochemical stainings revealed NPS-positive cells in the central and basolateral region of the amygdala in rAAV-NPS but not rAAV-Empty rats, indicating successful transduction. Our study provides novel evidence for sustained anxiolytic-like properties of NPS by transgenic overexpression. These data suggest that rAAV-NPS application deserves further attention as a potential treatment strategy for anxiety in humans.

Original languageEnglish
JournalBehavioural Brain Research
Volume367
Pages (from-to)28-34
Number of pages7
ISSN0166-4328
DOIs
Publication statusPublished - Jul 2019

Keywords

  • Adeno-associated virus
  • Anxiety
  • Depression
  • Neuropeptide S

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