Active Pumping in Endothelial Cell Tight Junctions

Research output: Contribution to conferencePosterResearch

Standard

Active Pumping in Endothelial Cell Tight Junctions. / Thrysøe, Samuel Alberg; Nygaard, Jens Vinge.

2011. Poster session presented at Biomechanics in vascular Biology and Cardiovascular Disease, Rotterdam, Netherlands.

Research output: Contribution to conferencePosterResearch

Harvard

Thrysøe, SA & Nygaard, JV 2011, 'Active Pumping in Endothelial Cell Tight Junctions', Biomechanics in vascular Biology and Cardiovascular Disease, Rotterdam, Netherlands, 14/04/2011 - 15/04/2011.

APA

Thrysøe, S. A., & Nygaard, J. V. (2011). Active Pumping in Endothelial Cell Tight Junctions. Poster session presented at Biomechanics in vascular Biology and Cardiovascular Disease, Rotterdam, Netherlands.

CBE

Thrysøe SA, Nygaard JV. 2011. Active Pumping in Endothelial Cell Tight Junctions. Poster session presented at Biomechanics in vascular Biology and Cardiovascular Disease, Rotterdam, Netherlands.

MLA

Thrysøe, Samuel Alberg and Jens Vinge Nygaard Active Pumping in Endothelial Cell Tight Junctions. Biomechanics in vascular Biology and Cardiovascular Disease, 14 Apr 2011, Rotterdam, Netherlands, Poster, 2011. 1 p.

Vancouver

Thrysøe SA, Nygaard JV. Active Pumping in Endothelial Cell Tight Junctions. 2011. Poster session presented at Biomechanics in vascular Biology and Cardiovascular Disease, Rotterdam, Netherlands.

Author

Thrysøe, Samuel Alberg ; Nygaard, Jens Vinge. / Active Pumping in Endothelial Cell Tight Junctions. Poster session presented at Biomechanics in vascular Biology and Cardiovascular Disease, Rotterdam, Netherlands.1 p.

Bibtex

@conference{2481cf6caa91494a80fe005b3447761b,
title = "Active Pumping in Endothelial Cell Tight Junctions",
abstract = "Biomechanical factors have long been associated with the initiation and development of atherosclerosis. However, this research has primarily been focused on the importance of Wall Shear Stress (WSS), which has been shown to be low or oscillating in areas prone to the development of atherosclerotic plaques.1 The emergence of finite element solvers capable of coupling different physics has allowed further investigations into the initiation of atherosclerosis. In this abstract, we present a novel method for coupling mechanical deformations of endothelial cells to the mass transport of lipid through tight junctions. ",
author = "Thrys{\o}e, {Samuel Alberg} and Nygaard, {Jens Vinge}",
year = "2011",
month = may,
language = "English",
note = "Biomechanics in vascular Biology and Cardiovascular Disease ; Conference date: 14-04-2011 Through 15-04-2011",

}

RIS

TY - CONF

T1 - Active Pumping in Endothelial Cell Tight Junctions

AU - Thrysøe, Samuel Alberg

AU - Nygaard, Jens Vinge

PY - 2011/5

Y1 - 2011/5

N2 - Biomechanical factors have long been associated with the initiation and development of atherosclerosis. However, this research has primarily been focused on the importance of Wall Shear Stress (WSS), which has been shown to be low or oscillating in areas prone to the development of atherosclerotic plaques.1 The emergence of finite element solvers capable of coupling different physics has allowed further investigations into the initiation of atherosclerosis. In this abstract, we present a novel method for coupling mechanical deformations of endothelial cells to the mass transport of lipid through tight junctions.

AB - Biomechanical factors have long been associated with the initiation and development of atherosclerosis. However, this research has primarily been focused on the importance of Wall Shear Stress (WSS), which has been shown to be low or oscillating in areas prone to the development of atherosclerotic plaques.1 The emergence of finite element solvers capable of coupling different physics has allowed further investigations into the initiation of atherosclerosis. In this abstract, we present a novel method for coupling mechanical deformations of endothelial cells to the mass transport of lipid through tight junctions.

M3 - Poster

T2 - Biomechanics in vascular Biology and Cardiovascular Disease

Y2 - 14 April 2011 through 15 April 2011

ER -