Signe Voigt Lauridsen

Coagulation Profile after Spontaneous Intracerebral Hemorrhage: A Cohort Study

Research output: Contribution to journal/Conference contribution in journal/Contribution to newspaperJournal articleResearchpeer-review

Standard

Coagulation Profile after Spontaneous Intracerebral Hemorrhage : A Cohort Study. / Lauridsen, Signe Voigt; Hvas, Anne-Mette; Sandgaard, Emilie et al.

In: Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association, Vol. 27, No. 11, 2018, p. 2951-2961.

Research output: Contribution to journal/Conference contribution in journal/Contribution to newspaperJournal articleResearchpeer-review

Harvard

Lauridsen, SV, Hvas, A-M, Sandgaard, E, Gyldenholm, T, Rahbek, C, Hjort, N, Tønnesen, EK & Hvas, CL 2018, 'Coagulation Profile after Spontaneous Intracerebral Hemorrhage: A Cohort Study', Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association, vol. 27, no. 11, pp. 2951-2961. https://doi.org/10.1016/j.jstrokecerebrovasdis.2018.06.022

APA

Lauridsen, S. V., Hvas, A-M., Sandgaard, E., Gyldenholm, T., Rahbek, C., Hjort, N., Tønnesen, E. K., & Hvas, C. L. (2018). Coagulation Profile after Spontaneous Intracerebral Hemorrhage: A Cohort Study. Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association, 27(11), 2951-2961. https://doi.org/10.1016/j.jstrokecerebrovasdis.2018.06.022

CBE

Lauridsen SV, Hvas A-M, Sandgaard E, Gyldenholm T, Rahbek C, Hjort N, Tønnesen EK, Hvas CL. 2018. Coagulation Profile after Spontaneous Intracerebral Hemorrhage: A Cohort Study. Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association. 27(11):2951-2961. https://doi.org/10.1016/j.jstrokecerebrovasdis.2018.06.022

MLA

Lauridsen, Signe Voigt et al. "Coagulation Profile after Spontaneous Intracerebral Hemorrhage: A Cohort Study". Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association. 2018, 27(11). 2951-2961. https://doi.org/10.1016/j.jstrokecerebrovasdis.2018.06.022

Vancouver

Lauridsen SV, Hvas A-M, Sandgaard E, Gyldenholm T, Rahbek C, Hjort N et al. Coagulation Profile after Spontaneous Intracerebral Hemorrhage: A Cohort Study. Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association. 2018;27(11):2951-2961. doi: 10.1016/j.jstrokecerebrovasdis.2018.06.022

Author

Lauridsen, Signe Voigt ; Hvas, Anne-Mette ; Sandgaard, Emilie et al. / Coagulation Profile after Spontaneous Intracerebral Hemorrhage : A Cohort Study. In: Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association. 2018 ; Vol. 27, No. 11. pp. 2951-2961.

Bibtex

@article{444ee11176b9477198326178e9e2f221,
title = "Coagulation Profile after Spontaneous Intracerebral Hemorrhage: A Cohort Study",
abstract = "Background: Intracerebral hemorrhage (ICH) causes death or disability and the incidence increases with age. Knowledge of acute hemostatic function in patients with ICH without anticoagulant and antiplatelet therapy is sparse. Increased knowledge of the coagulation profile in the acute phase of ICH could improve acute treatment and recovery. We investigated coagulation at admission and changes in coagulation during the first 24 hours after symptom onset. Methods: Enrolled were 41 ICH patients without anticoagulant or antiplatelet therapy admitted to Aarhus University Hospital, Denmark. Blood samples were collected at admission, 6, and 24 hours after symptom onset. Thromboelastometry (ROTEM), thrombin generation, and thrombin-antithrombin (TAT) complex were analyzed. Clinical outcome was evaluated using the National Institute of Health Stroke Scale, the Modified Rankin Score, and mortality. Results: At admission, compared with healthy individuals, ICH patients had increased maximum clot firmness (EXTEM P <.0001; INTEM P <.0001; FIBTEM P <.0001), increased platelet maximum clot elasticity (P <.0001) in ROTEM, higher peak thrombin (P <.0001) and endogenous thrombin potential (P =.01) in thrombin generation, and elevated TAT complex levels. During 24 hours after significantly, while thrombin generation showed decreased peak thrombin (P <.0001) and endogenous thrombin potential (P <.0001). Coagulation test results did not differ between patients when stratified according to clinical outcome. Conclusions: ICH patients without anticoagulant or antiplatelet therapy demonstrated activated coagulation at admission and within 24 hours after symptom onset.",
keywords = "Adult, Aged, Aged, 80 and over, Antithrombin III, Biomarkers/blood, Blood Coagulation, Case-Control Studies, Cerebral Hemorrhage/blood, Denmark, Disability Evaluation, Female, Hospitals, University, Humans, Male, Middle Aged, Patient Admission, Peptide Hydrolases/blood, Predictive Value of Tests, Prospective Studies, Thrombelastography, Thrombin/metabolism, Time Factors",
author = "Lauridsen, {Signe Voigt} and Anne-Mette Hvas and Emilie Sandgaard and Tua Gyldenholm and Christian Rahbek and Niels Hjort and T{\o}nnesen, {Else Kirstine} and Hvas, {Christine Lodberg}",
note = "Copyright {\textcopyright} 2018. Published by Elsevier Inc.",
year = "2018",
doi = "10.1016/j.jstrokecerebrovasdis.2018.06.022",
language = "English",
volume = "27",
pages = "2951--2961",
journal = "Journal of Stroke & Cerebrovascular Diseases",
issn = "1052-3057",
publisher = "W.B. Saunders Co.",
number = "11",

}

RIS

TY - JOUR

T1 - Coagulation Profile after Spontaneous Intracerebral Hemorrhage

T2 - A Cohort Study

AU - Lauridsen, Signe Voigt

AU - Hvas, Anne-Mette

AU - Sandgaard, Emilie

AU - Gyldenholm, Tua

AU - Rahbek, Christian

AU - Hjort, Niels

AU - Tønnesen, Else Kirstine

AU - Hvas, Christine Lodberg

N1 - Copyright © 2018. Published by Elsevier Inc.

PY - 2018

Y1 - 2018

N2 - Background: Intracerebral hemorrhage (ICH) causes death or disability and the incidence increases with age. Knowledge of acute hemostatic function in patients with ICH without anticoagulant and antiplatelet therapy is sparse. Increased knowledge of the coagulation profile in the acute phase of ICH could improve acute treatment and recovery. We investigated coagulation at admission and changes in coagulation during the first 24 hours after symptom onset. Methods: Enrolled were 41 ICH patients without anticoagulant or antiplatelet therapy admitted to Aarhus University Hospital, Denmark. Blood samples were collected at admission, 6, and 24 hours after symptom onset. Thromboelastometry (ROTEM), thrombin generation, and thrombin-antithrombin (TAT) complex were analyzed. Clinical outcome was evaluated using the National Institute of Health Stroke Scale, the Modified Rankin Score, and mortality. Results: At admission, compared with healthy individuals, ICH patients had increased maximum clot firmness (EXTEM P <.0001; INTEM P <.0001; FIBTEM P <.0001), increased platelet maximum clot elasticity (P <.0001) in ROTEM, higher peak thrombin (P <.0001) and endogenous thrombin potential (P =.01) in thrombin generation, and elevated TAT complex levels. During 24 hours after significantly, while thrombin generation showed decreased peak thrombin (P <.0001) and endogenous thrombin potential (P <.0001). Coagulation test results did not differ between patients when stratified according to clinical outcome. Conclusions: ICH patients without anticoagulant or antiplatelet therapy demonstrated activated coagulation at admission and within 24 hours after symptom onset.

AB - Background: Intracerebral hemorrhage (ICH) causes death or disability and the incidence increases with age. Knowledge of acute hemostatic function in patients with ICH without anticoagulant and antiplatelet therapy is sparse. Increased knowledge of the coagulation profile in the acute phase of ICH could improve acute treatment and recovery. We investigated coagulation at admission and changes in coagulation during the first 24 hours after symptom onset. Methods: Enrolled were 41 ICH patients without anticoagulant or antiplatelet therapy admitted to Aarhus University Hospital, Denmark. Blood samples were collected at admission, 6, and 24 hours after symptom onset. Thromboelastometry (ROTEM), thrombin generation, and thrombin-antithrombin (TAT) complex were analyzed. Clinical outcome was evaluated using the National Institute of Health Stroke Scale, the Modified Rankin Score, and mortality. Results: At admission, compared with healthy individuals, ICH patients had increased maximum clot firmness (EXTEM P <.0001; INTEM P <.0001; FIBTEM P <.0001), increased platelet maximum clot elasticity (P <.0001) in ROTEM, higher peak thrombin (P <.0001) and endogenous thrombin potential (P =.01) in thrombin generation, and elevated TAT complex levels. During 24 hours after significantly, while thrombin generation showed decreased peak thrombin (P <.0001) and endogenous thrombin potential (P <.0001). Coagulation test results did not differ between patients when stratified according to clinical outcome. Conclusions: ICH patients without anticoagulant or antiplatelet therapy demonstrated activated coagulation at admission and within 24 hours after symptom onset.

KW - Adult

KW - Aged

KW - Aged, 80 and over

KW - Antithrombin III

KW - Biomarkers/blood

KW - Blood Coagulation

KW - Case-Control Studies

KW - Cerebral Hemorrhage/blood

KW - Denmark

KW - Disability Evaluation

KW - Female

KW - Hospitals, University

KW - Humans

KW - Male

KW - Middle Aged

KW - Patient Admission

KW - Peptide Hydrolases/blood

KW - Predictive Value of Tests

KW - Prospective Studies

KW - Thrombelastography

KW - Thrombin/metabolism

KW - Time Factors

U2 - 10.1016/j.jstrokecerebrovasdis.2018.06.022

DO - 10.1016/j.jstrokecerebrovasdis.2018.06.022

M3 - Journal article

C2 - 30072172

VL - 27

SP - 2951

EP - 2961

JO - Journal of Stroke & Cerebrovascular Diseases

JF - Journal of Stroke & Cerebrovascular Diseases

SN - 1052-3057

IS - 11

ER -