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Lin, X, Li, J, Yin, G, Zhao, Q, Elias, D, Lykkesfeldt, AE, Stenvang, J, Brünner, N, Wang, J, Yang, H
, Bolund, L & Ditzel, HJ 2013, '
Integrative analyses of gene expression and DNA methylation profiles in breast cancer cell line models of tamoxifen-resistance indicate a potential role of cells with stem-like properties',
Breast Cancer Research (Online Edition), vol. 15, no. 6, pp. R119.
https://doi.org/10.1186/bcr3588APA
Lin, X., Li, J., Yin, G., Zhao, Q., Elias, D., Lykkesfeldt, A. E., Stenvang, J., Brünner, N., Wang, J., Yang, H.
, Bolund, L., & Ditzel, H. J. (2013).
Integrative analyses of gene expression and DNA methylation profiles in breast cancer cell line models of tamoxifen-resistance indicate a potential role of cells with stem-like properties.
Breast Cancer Research (Online Edition),
15(6), R119.
https://doi.org/10.1186/bcr3588CBE
Lin X, Li J, Yin G, Zhao Q, Elias D, Lykkesfeldt AE, Stenvang J, Brünner N, Wang J, Yang H, et al. 2013.
Integrative analyses of gene expression and DNA methylation profiles in breast cancer cell line models of tamoxifen-resistance indicate a potential role of cells with stem-like properties.
Breast Cancer Research (Online Edition). 15(6):R119.
https://doi.org/10.1186/bcr3588MLA
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Bibtex
@article{50b26332c3494d45b98ed120140d31c6,
title = "Integrative analyses of gene expression and DNA methylation profiles in breast cancer cell line models of tamoxifen-resistance indicate a potential role of cells with stem-like properties",
abstract = "Development of resistance to tamoxifen is an important clinical issue in the treatment of breast cancer. Tamoxifen resistance may be the result of acquisition of epigenetic regulation within breast cancer cells, such as DNA methylation, resulting in changed mRNA expression of genes pivotal for estrogen-dependent growth. Alternatively, tamoxifen resistance may be due to selection of pre-existing resistant cells, or a combination of the two mechanisms.",
author = "Xue Lin and Jian Li and Guangliang Yin and Qian Zhao and Daniel Elias and Lykkesfeldt, {Anne E} and Jan Stenvang and Nils Br{\"u}nner and Jun Wang and Huanming Yang and Lars Bolund and Ditzel, {Henrik J}",
year = "2013",
month = dec,
day = "19",
doi = "10.1186/bcr3588",
language = "English",
volume = "15",
pages = "R119",
journal = "Breast Cancer Research (Online Edition)",
issn = "1465-5411",
publisher = "BioMed Central Ltd.",
number = "6",
}
RIS
TY - JOUR
T1 - Integrative analyses of gene expression and DNA methylation profiles in breast cancer cell line models of tamoxifen-resistance indicate a potential role of cells with stem-like properties
AU - Lin, Xue
AU - Li, Jian
AU - Yin, Guangliang
AU - Zhao, Qian
AU - Elias, Daniel
AU - Lykkesfeldt, Anne E
AU - Stenvang, Jan
AU - Brünner, Nils
AU - Wang, Jun
AU - Yang, Huanming
AU - Bolund, Lars
AU - Ditzel, Henrik J
PY - 2013/12/19
Y1 - 2013/12/19
N2 - Development of resistance to tamoxifen is an important clinical issue in the treatment of breast cancer. Tamoxifen resistance may be the result of acquisition of epigenetic regulation within breast cancer cells, such as DNA methylation, resulting in changed mRNA expression of genes pivotal for estrogen-dependent growth. Alternatively, tamoxifen resistance may be due to selection of pre-existing resistant cells, or a combination of the two mechanisms.
AB - Development of resistance to tamoxifen is an important clinical issue in the treatment of breast cancer. Tamoxifen resistance may be the result of acquisition of epigenetic regulation within breast cancer cells, such as DNA methylation, resulting in changed mRNA expression of genes pivotal for estrogen-dependent growth. Alternatively, tamoxifen resistance may be due to selection of pre-existing resistant cells, or a combination of the two mechanisms.
U2 - 10.1186/bcr3588
DO - 10.1186/bcr3588
M3 - Journal article
C2 - 24355041
VL - 15
SP - R119
JO - Breast Cancer Research (Online Edition)
JF - Breast Cancer Research (Online Edition)
SN - 1465-5411
IS - 6
ER -