Research output: Contribution to journal/Conference contribution in journal/Contribution to newspaper › Journal article › Research › peer-review
Susceptibility to Leprosy is Associated with M-ficolin Polymorphisms. / Boldt, Angelica B W; Sanchez, Maria Iolanda N; Stahlke, Ewalda R S; Steffensen, Rudi; Thiel, Steffen; Jensenius, Jens C; Prevedello, Flávia Costa; Mira, Marcelo Távora; Kun, Jürgen F J; Messias-Reason, Iara J T.
In: Journal of Clinical Immunology, Vol. 33, No. 1, 2013, p. 210-9.Research output: Contribution to journal/Conference contribution in journal/Contribution to newspaper › Journal article › Research › peer-review
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TY - JOUR
T1 - Susceptibility to Leprosy is Associated with M-ficolin Polymorphisms
AU - Boldt, Angelica B W
AU - Sanchez, Maria Iolanda N
AU - Stahlke, Ewalda R S
AU - Steffensen, Rudi
AU - Thiel, Steffen
AU - Jensenius, Jens C
AU - Prevedello, Flávia Costa
AU - Mira, Marcelo Távora
AU - Kun, Jürgen F J
AU - Messias-Reason, Iara J T
PY - 2013
Y1 - 2013
N2 - Mycobacterium leprae exploits complement activation and opsonophagocytosis to infect phagocytes. M-ficolin is encoded by the FCN1 gene and initiates the lectin pathway on monocyte surfaces. We investigated FCN1 promoter polymorphisms that could be responsible for the high interindividual variability of M-ficolin levels and for modulating leprosy susceptibility.
AB - Mycobacterium leprae exploits complement activation and opsonophagocytosis to infect phagocytes. M-ficolin is encoded by the FCN1 gene and initiates the lectin pathway on monocyte surfaces. We investigated FCN1 promoter polymorphisms that could be responsible for the high interindividual variability of M-ficolin levels and for modulating leprosy susceptibility.
U2 - 10.1007/s10875-012-9770-4
DO - 10.1007/s10875-012-9770-4
M3 - Journal article
C2 - 22941510
VL - 33
SP - 210
EP - 219
JO - Journal of Clinical Immunology
JF - Journal of Clinical Immunology
SN - 0271-9142
IS - 1
ER -