Molecular strategies to inhibit HIV-1 replication

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Molecular strategies to inhibit HIV-1 replication. / Nielsen, Morten Hjuler; Pedersen, Finn Skou; Kjems, Jørgen.

I: Retrovirology, Bind 2, 16.02.2005, s. 10.

Publikation: Bidrag til tidsskrift/Konferencebidrag i tidsskrift /Bidrag til avisTidsskriftartikelForskningpeer review

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Nielsen, Morten Hjuler ; Pedersen, Finn Skou ; Kjems, Jørgen. / Molecular strategies to inhibit HIV-1 replication. I: Retrovirology. 2005 ; Bind 2. s. 10.

Bibtex

@article{3974d1dd03034987b9b58007e3ffeb71,
title = "Molecular strategies to inhibit HIV-1 replication",
abstract = "The human immunodeficiency virus type 1 (HIV-1) is the primary cause of the acquired immunodeficiency syndrome (AIDS), which is a slow, progressive and degenerative disease of the human immune system. The pathogenesis of HIV-1 is complex and characterized by the interplay of both viral and host factors. An intense global research effort into understanding the individual steps of the viral replication cycle and the dynamics during an infection has inspired researchers in the development of a wide spectrum of antiviral strategies. Practically every stage in the viral life cycle and every viral gene product is a potential target. In addition, several strategies are targeting host proteins that play an essential role in the viral life cycle. This review summarizes the main genetic approaches taken in such antiviral strategies.",
keywords = "Antiviral Agents, HIV-1, Humans, RNA Interference, RNA, Antisense, RNA, Messenger, Viral Proteins, Virus Replication",
author = "Nielsen, {Morten Hjuler} and Pedersen, {Finn Skou} and J{\o}rgen Kjems",
year = "2005",
month = feb,
day = "16",
doi = "10.1186/1742-4690-2-10",
language = "English",
volume = "2",
pages = "10",
journal = "Retrovirology",
issn = "1742-4690",
publisher = "BioMed Central",

}

RIS

TY - JOUR

T1 - Molecular strategies to inhibit HIV-1 replication

AU - Nielsen, Morten Hjuler

AU - Pedersen, Finn Skou

AU - Kjems, Jørgen

PY - 2005/2/16

Y1 - 2005/2/16

N2 - The human immunodeficiency virus type 1 (HIV-1) is the primary cause of the acquired immunodeficiency syndrome (AIDS), which is a slow, progressive and degenerative disease of the human immune system. The pathogenesis of HIV-1 is complex and characterized by the interplay of both viral and host factors. An intense global research effort into understanding the individual steps of the viral replication cycle and the dynamics during an infection has inspired researchers in the development of a wide spectrum of antiviral strategies. Practically every stage in the viral life cycle and every viral gene product is a potential target. In addition, several strategies are targeting host proteins that play an essential role in the viral life cycle. This review summarizes the main genetic approaches taken in such antiviral strategies.

AB - The human immunodeficiency virus type 1 (HIV-1) is the primary cause of the acquired immunodeficiency syndrome (AIDS), which is a slow, progressive and degenerative disease of the human immune system. The pathogenesis of HIV-1 is complex and characterized by the interplay of both viral and host factors. An intense global research effort into understanding the individual steps of the viral replication cycle and the dynamics during an infection has inspired researchers in the development of a wide spectrum of antiviral strategies. Practically every stage in the viral life cycle and every viral gene product is a potential target. In addition, several strategies are targeting host proteins that play an essential role in the viral life cycle. This review summarizes the main genetic approaches taken in such antiviral strategies.

KW - Antiviral Agents

KW - HIV-1

KW - Humans

KW - RNA Interference

KW - RNA, Antisense

KW - RNA, Messenger

KW - Viral Proteins

KW - Virus Replication

U2 - 10.1186/1742-4690-2-10

DO - 10.1186/1742-4690-2-10

M3 - Journal article

C2 - 15715913

VL - 2

SP - 10

JO - Retrovirology

JF - Retrovirology

SN - 1742-4690

ER -