Aarhus Universitets segl

English

Du er her: AU » Om AU » Organisation » Jens Christian Jensenius » Publikationer » Genetically-defined deficiency of mannose-binding lectin ...

Om AU

Jens Christian Jensenius

Genetically-defined deficiency of mannose-binding lectin is associated with protection after experimental stroke in mice and outcome in human stroke

Publikation: Bidrag til tidsskrift/Konferencebidrag i tidsskrift /Bidrag til avis › Tidsskriftartikel › Forskning › peer review

DOI

  • Alvaro Cervera
    • ,
  • Anna M Planas
    • ,
  • Carles Justicia
    • ,
  • Xabier Urra
    • ,
  • Jens Christian Jensenius
  • Ferran Torres
    • ,
  • Francisco Lozano
    • ,
  • Angel Chamorro
The complement system is a major effector of innate immunity that has been involved in stroke brain damage. Complement activation occurs through the classical, alternative and lectin pathways. The latter is initiated by mannose-binding lectin (MBL) and MBL-associated serine proteases (MASPs). Here we investigated whether the lectin pathway contributes to stroke outcome in mice and humans.
OriginalsprogEngelsk
TidsskriftP L o S One
Vol/bind5
Nummer2
Sider (fra-til)e8433
ISSN1932-6203
DOI
StatusUdgivet - 2010

Se relationer på Aarhus Universitet Citationsformater

ID: 43925499