Corneal dystrophy mutations drive pathogenesis by targeting TGFBIp stability and solubility in a latent amyloid-forming domain

Research output: Contribution to journal/Conference contribution in journal/Contribution to newspaperJournal articleResearchpeer-review

Original languageEnglish
JournalJournal of Molecular Biology
Volume430
Issue number8
Pages (from-to)1116-1140
ISSN0022-2836
DOIs
StatePublished - 13 Apr 2018

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